Steroid responsive meningitis

Steroid Responsive Meningitis - Samantha Goldberg BVSc MRCVS

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The Veterinary Record, April 7th 1973--Polyarteritis in the Dog: A Case Report   Dr. Roughie’s Questions and Answers- Steroid Responsive Meningitis-Vasculitis: The Disease With Many Names   Kasmin D. Bittle DVM   /images/Health/Dr-Roughie-ST-Column/
Neck Pain By Dr. J. E. Dillberger,  /images/Health/Dr-Roughie-ST-Column/

Cells of the zona fasciculata and zona reticularis lack aldosterone synthase (CYP11B2) that converts corticosterone to aldosterone, and thus these tissues produce only the weak mineralocorticoid corticosterone. However, both these zones do contain the CYP17A1 missing in zona glomerulosa and thus produce the major glucocorticoid, cortisol. Zona fasciculata and zona reticularis cells also contain CYP17A1, whose 17,20-lyase activity is responsible for producing the androgens, dehydroepiandrosterone (DHEA) and androstenedione. Thus, fasciculata and reticularis cells can make corticosteroids and the adrenal androgens, but not aldosterone.

Oral cyclosporine was effective in the DEBR model for alopecia areata. All rats had a full pelage by 5 weeks of treatment with 10 mg/kg/d, 5 d/wk for 7 weeks. Studies in humans also have proven efficacy with doses of 6 mg/kg/d for 3 months in 6 patients. All patients experienced regrowth, and cosmetically acceptable regrowth was seen in 3 of 6 patients. Unfortunately, all patients relapsed within 3 months of discontinuation of cyclosporine. No evidence indicates that CsA can prevent hair loss during an active episode because reports have described patients taking CsA who developed alopecia areata while they were under treatment for unrelated conditions.

Steroid responsive meningitis

steroid responsive meningitis

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